So you think… but it’s not clear how mutations translate into phenotypic change.
If need triggers a more efficient search, then organisms will have a better chance of getting what they need.
End of the day, scientists are still unravelling the complexities of genotypes, phenotypes, the various ways the cell “writes” into the genome etc.
A conclusion of “randomness” could be based on ignorance.
Not at all… the goal is not just a particular mutation. It’s a phenotype that gives gain of function.
Some types of mutations might bring about bigger changes in phenotype than others.
And phenotypes exist on the organisms level.
No “search” can be random… faster/more efficient searches can find a solution where other searches cannot… the impact is consistent, not random.
This is why the immune system is successful.
Before you were questioning if mutations even changed phenotypes.
That’s a bold claim with zero evidence to back it.
If mutations are not a random search, then why do these mutation processes also produce neutral and detrimental effects? Why do only a tiny, tiny percentage of organisms get the needed mutation in a given environment?
Unless we don’t take the word to indicate a “desire to find”. Which we can do, because we don’t like to get hung up on particular words, what matters are concepts.
Humans are biological systems, and we desire things. The point I am making is that the processes that produce mutations are blind to what will help or hurt the organism.
there is a different between a simple trait and a complex one. and the flagellum is a complex one. im not sure that the same is true for the antifreeze protein.