That’s just you making an excuse to avoid answering. And whatever your real position may be, your expressed positions are highly confused.
You should at least end a question with a question mark. And I see you’re still avoiding any real answers.
You clearly don’t understand the scientific method. Kepler was able to come up with his laws of planetary motion without knowing anything about universal gravitation. Newton was able to come up with his law of universal gravitation without knowing anything about the Higgs boson. And so on. Science always works on incomplete information. You’re just using the common creationist excuse that if we don’t know everything we therefore know nothing. Not true. We have copious evidence of common descent that doesn’t depend on knowing how mutations happen. And anyway we do know how mutations happen. You are just clinging to the notion that not enough of them happen, or not enough of them get fixed. But you have no evidence for that, and your unsupported claim contradicts the historical evidence. If you were actually interested in science, you would have noticed that.
Please explain. Why does that make it a poor alternative? And what do you mean by “muddles” here?
In the first case, that’s just an admission that the chromosome evidence is not relevant, since you need something else instead. In the second case, since some of them are fixed in their local populations (some of them isolated, in fact), that’s more than variation in a population. Further, all that’s needed is that the variants attain a high frequency, which shows that they could become fixed, i.e. that they are not strongly deleterious. These are nothing but excuses to ignore the data.
What evidence do you have of this mechanism? And when did the argument become about gain and loss of function? This is the first time that subject has been mentioned.
The logic is quite simple. We have a phylogenetic tree that puts flying tinamous in the middle of the flightless ratites. Either flight was lost and regained, or it was lost multiple times. We know of a great many cases in which flight was lost in birds (mostly island rails), none in which it was regained. Notice that there’s nothing at all about why or how it was lost. Though you could certainly come up with hypotheses about that, I didn’t in that paper. See? No assumptions about natural variation, and the phylogeny is prior to any inference of events, regardless of their cause.
That’s right. You are determined not to. The basis is that nested hierarchy or, if you prefer, the fit of the data to a particular tree. There’s more than that, data of various other sorts, but that’s what I usually talk about.
Actually, we do. There are variations in gene presence and absence within populations. And between populations, some genes are clearly homologous to non-genic sequences in other populations. What explanation other than alteration of a non-gene into a gene would produce such a thing?
Would I really have to? Is it not exceedingly obvious, if you just think about it for a few moments? Start with a branching tree of species. Now sprinkle changes randomly over that tree. Isn’t it clear that the distribution of characterstics would follow a nested hierarchy, such that you could reconstruct the tree based on that distribution? And it’s even been done in the laboratory, with virus evolution, and the true relationships were reconstructed based on that nested hierarchy of sequence data. How can anyone fail to understand that after so many years?
Better question: why, when we’re talking about one thing, do you change the subject to another?
More deflection.
Great. So we’re all done?
You misunderstand the degree of Bill’s confusion. When he says “gene arrangements” he means the sets of genes that different species possess, not their arrangement. He uses words differently from other people.