Methinks it is sort-of like two weasels

Mentioning it, but not including it in any of his simplistic calculations. Interestingly, you completely contradicted this claim in the other thread:

No, you are misrepresenting the paper. Did they assay all the mutants known in 2014? Have other mutants been identified since then?

Convenient. There’s a search function at the top of the page.

Your claim is spectacularly, totally, objectively false. Why do you make such claims?

Mentioning it once doesn’t constitute including it in his simplistic calculations.

Isn’t that precisely what happens in the phases of the Plasmodium life cycle in mosquitoes and rats? Is the fitness of these mutants higher or lower than wild-type in the absence of chloroquine, Lee?

Please show me where anything resembling that is included in Behe’s calculation.

Because they create variation without new mutations.

Why do Summers et al. refer to haplotypes, but Behe never uses the word in the book? That should tell you something.

Why does Behe never mention epistasis?

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