Some Unresolved Issues about Properly Understanding Genetic Entropy

[Mercer (John Mercer)]

@thoughtful

You’re missing the point.

I don’t think so. This comes back to his pointing out that the mutational and evolutionary mechanisms we observe in real time account for these differences.

You said 11 days ago that “the evidence isn’t actually consistent with that.”

Two days ago, you wrote, “I definitely could have provided data in the examples you mentioned.”

This is a variation on one of those examples I mentioned. I don’t see any reason why you could have provided data 11 days ago, but are unable to today.

@thoughtful

We don’t share a common ancestor with mice.

Evidence?

@thoughtful

Ok, I will have to do better in the future by providing more data to show you that I have examined the data.

You’re failing.

Today is the future relative to two days ago. How about providing any data, much less more? You’ve made multiple assertions here with zero evidence.

@thoughtful

Random mutation + selection can’t create.

Yet the evidence shows that those two mechanisms create all sorts of incredibly specific antibodies in your body in less than two weeks. If that isn’t true, how does the SARS-CoV-2 vaccine work in the YEC universe?

@thoughtful

It seems you don’t want to be specific with examples or data.

I don’t know if “you” was singular or plural, but Taq opened with specific examples and data:

@T_aquaticus

The >90% of the human genome that is not under selection has very little similarity to the mouse genome. Mice and humans are doing just fine despite all of these mutations. This is in comparison to shared regions that are under selection which have up to 85% similarity between humans and mice.

That is evidence, Valerie. You don’t offer any.