Wrong. All we know is that is a very rough guesstimate made by one author in one paper of how often this resistance has arisen in human subjects being treated for malaria. In another paper the same author uses the number 1 in 10 19. Maybe rats are teeming with resistant parasites that we know nothing about.
Now, this raises an interesting question. Your reasoning here seems more or less sound, and as it happens CQR does exist for vivax, but is much less common than for falciparum.
Here is the question: If Behe had written his book about CQR in vivax rather than in falciparum, would he not have defined this as the “edge of evolution”? I can’t see why not. But then, following his reasoning, CQR should not be more common in falciparum, because this would require that it occur beyond Behe’s “edge.”
And yet it is.
Can you explain this finding that should be impossible if Behe’s reasoning is correct?