Continuing the discussion from Gil's testable ID hypothesis:
I’m not sure how that works in your mind, but…
Let’s look at your attempt to cherry-pick, Bill. In the “Why is it hard to estimate FI” thread, you wrote:
Conveniently, the sarcomere is one of my areas of expertise.
You attempted to cherry-pick by citing actin:
Let me remind you of alpha actin which is part of an irreducible complex structure.
Mice have 5 generations per year and this split was at least 50 million years ago. At 50 mutations per generation that’s 10 billion neutral changes fixed in the population. There is real substantial functional constraint going on here.
There is. However, constraint is not information. Constraint as a proxy for function is the hypothesis that according to you, should be worked on.
So, Bill, please calculate @gpuccio’s “FI” for actin. Then together, we’ll do so for the alpha- and beta-cardiac myosin heavy chains so that we can see which has more “FI” and which has more functional information.